Date of Award

12-2019

Degree Type

Thesis

Degree Name

Master of Science

Department

Biology

Program

Biology (MS)

First Advisor/Chairperson

Dr Erich Ottem

Abstract

Skeletal muscle is a cytokine-producing organ capable of expressing various myokines, including tumor necrosis factor alpha, IL-6, IL-8, IL-15, IL-17, and brain-derived neurotrophic factor (BDNF) (Pederson, 2011, So et al., 2014, Schnyder and Handschin, 2015). BDNF is a small neurotrophic protein, that regulates neuron differentiation, maturation, and survival, and modulates central nervous system neurotransmitters (Oppenheim, 1991; Acheson et al., 1995; Binder and Scharfman, 2004; Reichardt, 2006). However, the role of BDNF in skeletal muscle health and maintenance is not well understood. To investigate this, we used a mouse model with Cre/Lox targeted gene deletion driven by the human alpha skeletal actin promoter, resulting in experimental animals with reduced or absent muscle-derived BDNF (MuscleBDNF+/-, MuscleBDNF-/-). We examined gastrocnemius and soleus muscles across multiple age groups (30, 90, 120, 180, and 210 days), assessing myofiber health in the absence or reduction of skeletal-derived BDNF. Significant pathology was observed in gastrocnemius tissues of both young and young-adult transgenic mice lacking endogenous BDNF, including mispositioned nuclei, split fibers, and fewer total myofibers, further histological analysis showed that fast-twitch myofibers were disproportionally affected. We next assessed the structure of gastrocnemius associated neuromuscular junctions of 120d mice and found that there is a presynaptic decrease of vesicle acetylcholine transporter and an increased probability of junction fragmentation in muscle BDNF knockout mice. Taken altogether these findings suggest that muscle-synthesized BDNF plays a role in the maintenance of muscle fibers and retrogradely affects innervating gastrocnemius associated motorneurons at the neuromuscular junction.

Access Type

NMU Users Only

Available for download on Wednesday, July 11, 2029

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